This is Dr. Fuhrman reporting from Seoul, South Korea. Physicians here are just as unaware about nutrition as they are in the United States. The difference is they are enthusiastic to learn more about nutritional science and how they can incorporate the most advanced discoveries into their practice.
I am spending this week in Korea conducting seminars for diverse group of physicians and health professionals in various specialties. Doing lots of bowing, shaking hands, taking photos, signing autographs and jumping on tables.
It’s fun to watch a room full of physicians chant in unison, "GOMBS!"
More to come from Korea!
Eating a high-nutrient diet focused on whole plant foods allows us to avoid many of the common environmental toxins and carcinogens that humans come into contact with on a regular basis, like heterocyclic amines, acrylamide, dioxins, and mercury. But there are other environmental carcinogens that do not come from the diet, and we need to be aware of these too.
The World Health Organization’s International Agency for Research on Cancer (IARC) issued a cautionary statement in 2011 about the potential dangers of radiation from radiofrequency electromagnetic fields, the type of radiation emitted by mobile phones and other wireless communication devices, radar, microwaves, radio, and television. A panel of scientists reviewed the existing research on the topic, and they decided to classify radiofrequency electromagnetic fields as “possibly carcinogenic to humans."1
The research is not conclusive yet, but the IARC advises caution while we wait for further studies to be done. Many of the studies so far have not found a connection between mobile phone use and cancer, but a few studies have found significant risk of brain tumors:
- The 13-country industry-funded Interphone study whose results were published last year did not find any increase in brain tumor risk associated with mobile phone use overall. However, they did see an increased risk of one type of brain tumor (glioma) at the highest exposure levels to radiofrequency electromagnetic fields.2
- An Israeli study found that greater cell phone use was associated with the development of parotid gland (a salivary gland) tumors.3
- A series of studies in Sweden have found links between cell phone use and brain tumors, particularly with long-term cell phone use (10 years or more) and in those who began using cell phones before age 20.4-6
- A review of 11 studies on cell phone use and brain tumors reported that most studies did not find associations, but 3 studies of long-term cell phone use did find that the risk of one type of brain tumor (acoustic neuroma) more than doubled in those who had used cell phones for 10 years or more.7
- A recent study found that an active cell phone against the ear increases glucose metabolism in the area of the brain closest to the antenna. Whether this has any relevance to the brain tumor studies is unknown, but this result does confirm that cell phone radiation can affect biological processes in the brain.8
Avoiding mobile phones is simply not an option for most of us, so what can we do to reduce our exposure?
The Environmental Working Group provides these tips:
- Choose a phone that emits lower levels of radiation – check the guide to the best and worst phones to see where your current phone ranks.
- Use a headset or speakerphone – holding your phone away from the body reduces radiation exposure significantly.
- Send a text message instead of calling when possible.
- Keep your cell phone turned off until you need to use it.
- Don’t talk when your phone is “working” to acquire a signal: when signal is low, or when you are in a moving vehicle.
- Children’s cell phone use should be very limited. Remember that children’s smaller bodies and dividing cells are more susceptible to carcinogenic influences.
Cell phones have only been in widespread use for a relatively short time, and it can take years or even decades for cancer to develop after exposure to a carcinogenic influence. We simply don’t know how safe cell phones are, and how their radiation may affect our physiology in the long term. In light of the data so far, cautious and conservative cell phone use is likely a valuable addition to an anti-cancer lifestyle.
1. Cell Phones Possibly Carcinogenic, WHO Says. Medscape Family Medicine News. http://www.medscape.com/viewarticle/743673?sssdmh=dm1.691945&src=nl_newsalert. Accessed June 22, 2011.
2. Brain tumour risk in relation to mobile telephone use: results of the INTERPHONE international case-control study. Int J Epidemiol 2010;39:675-694.
3. Sadetzki S, Chetrit A, Jarus-Hakak A, et al: Cellular phone use and risk of benign and malignant parotid gland tumors--a nationwide case-control study. Am J Epidemiol 2008;167:457-467.
4. Hardell L, Carlberg M: Mobile phones, cordless phones and the risk for brain tumours. Int J Oncol 2009;35:5-17.
5. Hardell L, Carlberg M, Hansson Mild K: Pooled analysis of case-control studies on malignant brain tumours and the use of mobile and cordless phones including living and deceased subjects. Int J Oncol 2011;38:1465-1474.
6. Hardell L, Carlberg M, Soderqvist F, et al: Meta-analysis of long-term mobile phone use and the association with brain tumours. Int J Oncol 2008;32:1097-1103.
7. Han YY, Kano H, Davis DL, et al: Cell phone use and acoustic neuroma: the need for standardized questionnaires and access to industry data. Surg Neurol 2009;72:216-222; discussion 222.
8. Volkow ND, Tomasi D, Wang GJ, et al: Effects of cell phone radiofrequency signal exposure on brain glucose metabolism. JAMA 2011;305:808-813.
Yesterday I ended six weeks of continual passive motion (CPM) therapy for a knee repair that I had back on June 3rd. Nothing has ever pushed my buttons of frustration more than this unexpected time of rehab, and I’ve experienced a lot of frustrating and stressful moments in my life up to this point. Perhaps it was because I felt well, and lying on my back with a leg strapped to a machine for that long during nice weather just felt so unnatural and claustrophobic. Perhaps I didn’t like being dependant upon others; or isolated from society; or unable to cook, clean, pull weeds, or do laundry the way I like. For whatever reason, the volcano of frustration increased with each passing day, and last week it erupted. Big time.
So I took off for the kitchen.
I had no idea what I was going after, but I was determined to find something.
After opening the cupboard doors and spotting a canister of cocoa powder I found my drug of choice. Now mind you, there’s absolutely nothing unhealthy about cocoa powder, but I wasn’t going after nutrients to nourish my body. I wasn’t hungry whatsoever.
I was mad.
I was frustrated AND desperately wanted a quick fix to soothe and pacify my frazzled nerves.
First I dumped a bunch of cocoa in with frozen bananas and made a scrumptious chocolate dessert. After that vanished I dumped a bunch more into some hot oatmeal. Ahhhh. I was soothed.
Or so I thought.
That was, until the next morning when I woke up. I had created not only a toxic headache, but stirred up even more frustrations so I headed straight back to the kitchen. This time I dumped at least twice as much of the powdery stuff into a frozen banana dessert and bowl of oatmeal. As I was in the process of bingeing, I gasped slightly at the amount of cocoa that I was putting into my body, but it didn’t bother me enough to quit.
However afterwards, when I came to my senses, I couldn’t believe that I had this much “attack” still left in me after almost three years of overcoming many emotional eating hurdles. Then the light bulb turned on. Have you ever misplaced an infant or toddler’s favorite pacifier or blanket? Watch out.
Immediately after that revelation I had absolutely no desire whatsoever to go after the cocoa powder. Why the change?
We all know that most everyone turns to food (and/or drinks) to celebrate happy occasions. Likewise, for years I’d viewed eating as a normal response to life’s stress, because it seemed like most everyone also turned to food as an acceptable drug of choice when frustrated too. Now I view it for what it truly is; silly and immature behavior, an out-of-control temper tantrum that only adds more stress.
I never had a pacifier, but I clearly remember the day my favorite blanket named “Pinky” was put on the burn pile. (I grew up on a farm.) I was six or seven-years-old and the blanket was in shambles. The time had come for me to let go of it; it was time for me to grow up.
Perhaps pacifying emotions through unhealthy eating behaviors is something that many of us need to let go of. Perhaps it’s time to grow up.
image credit: flickr by ff137
Acne is the most common skin condition in the U.S. About 85% of people in the Western world experience acne during their teenage years, but it can occur at any age. Acne is more than just pimples - it can leave permanent scars, and in many people, acne (even if it is not severe) can seriously affect quality of life, causing low self-esteem, withdrawal from social situations, anxiety, and depression.1
What causes acne?
There are four major components of acne: excessive production of oil by the skin, skin cells dividing excessively (hyperproliferation), bacteria, and inflammation.2 A pimple or lesion forms when a pore in the skin begins to clog with old, dead skin cells. Usually these cells are simply shed from the surface of the skin, but if too much oil is being produced, the dead cells can stick together and become trapped inside the pore. Bacteria also play a role – they can grow and multiply inside the pore, resulting in an inflammatory response.1
Does what we eat really affect acne?
For years doctors have proclaimed that diet has nothing to do with acne. That reflects the nutritional ignorance of physicians and their inexperience in treating disease with superior diet. Scientific studies have demonstrated that the diet is very important, because what we eat can affect the hormones that contribute to the oil production, hyperproliferation, and inflammation that cause acne. The acne-promoting dietary factors that have been most extensively studied are dairy products and high glycemic load foods – these factors influence hormonal (increase IGF-1 levels) and inflammatory factors increasing acne prevalence and severity.3,4
IGF-1: an important hormone that influences acne
Hormonal influences that affect insulin-like growth factor 1 (IGF-1) levels are key.5 Elevated IGF-1 levels lead to changes in gene expression that cause inflammation, hormonal changes, increased oil production, and development of acne lesions. Protein intake is the major factor that determines circulating IGF-1 levels, especially protein from dairy products. Read more in Dr. Fuhrman’s Healthy Times Newsletter on IGF-1.
A three-year prospective study of 9-15 year old girls found a 20% increase in acne prevalence in girls that had 2 or more servings of milk per day compared to less than 1 per week. This association held true for total, whole, low fat, and skim milk.6 The same researchers found a similar association in boys who drank skim milk (milk highest in protein).7 Furthermore, in the Nurses’ Health Study, dairy products eaten during high school were associated with acne during women’s teenage years.8
High glycemic load foods
Glycemic load (GL) is a measure of the effect of a certain food on blood glucose levels. High-GL foods like refined carbohydrates produce dangerous spikes in blood glucose, leading to excessive insulin levels in the blood (hyperinsulinemia), which contribute to diabetes, heart disease, and several cancers.9,10 Hyperinsulinemia not only promotes inflammation but also raises IGF-1 levels, further contributing to acne. A low glycemic load diet has been shown to improve acne symptoms, and decrease IGF-1 and skin oil production in several studies.11-13
Blood levels of zinc, carotenoids, and Vitamin E are known to be lower in acne patients compared to those without acne, suggesting that maintaining micronutrient adequacy may help to prevent acne.14,15 Carotenoids are abundant in green and orange vegetables, and vitamin E is abundant in nuts and seeds. Although pumpkin seeds and hemp seeds are rich in zinc, zinc absorption efficiency may be low on a plant-based diet, so a multivitamin and mineral supplement is recommended to assure optimal levels of zinc, iodine, Vitamin D and B12.
1. American Academy of Dermatology: Acne. http://www.aad.org/skin-conditions/dermatology-a-to-z/acne. Accessed June 29, 2011.
2. Costa A, Lage D, Moises TA: Acne and diet: truth or myth? An Bras Dermatol 2010;85:346-353.
3. Ferdowsian HR, Levin S: Does diet really affect acne? Skin Therapy Lett 2010;15:1-2, 5.
4. Melnik BC, Schmitz G: Role of insulin, insulin-like growth factor-1, hyperglycaemic food and milk consumption in the pathogenesis of acne vulgaris. Exp Dermatol 2009;18:833-841.
5. Danby FW: Diet and acne. Clin Dermatol 2008;26:93-96.
6. Adebamowo CA, Spiegelman D, Berkey CS, et al: Milk consumption and acne in adolescent girls. Dermatol Online J 2006;12:1.
7. Adebamowo CA, Spiegelman D, Berkey CS, et al: Milk consumption and acne in teenaged boys. J Am Acad Dermatol 2008;58:787-793.
8. Adebamowo CA, Spiegelman D, Danby FW, et al: High school dietary dairy intake and teenage acne. J Am Acad Dermatol 2005;52:207-214.
9. Barclay AW, Petocz P, McMillan-Price J, et al: Glycemic index, glycemic load, and chronic disease risk--a meta-analysis of observational studies. Am J Clin Nutr 2008;87:627-637.
10. Gnagnarella P, Gandini S, La Vecchia C, et al: Glycemic index, glycemic load, and cancer risk: a meta-analysis. Am J Clin Nutr 2008;87:1793-1801.
11. Smith R, Mann N, Makelainen H, et al: A pilot study to determine the short-term effects of a low glycemic load diet on hormonal markers of acne: a nonrandomized, parallel, controlled feeding trial. Mol Nutr Food Res 2008;52:718-726.
12. Smith RN, Braue A, Varigos GA, et al: The effect of a low glycemic load diet on acne vulgaris and the fatty acid composition of skin surface triglycerides. J Dermatol Sci 2008;50:41-52.
13. Smith RN, Mann NJ, Braue A, et al: A low-glycemic-load diet improves symptoms in acne vulgaris patients: a randomized controlled trial. Am J Clin Nutr 2007;86:107-115.
14. El-Akawi Z, Abdel-Latif N, Abdul-Razzak K: Does the plasma level of vitamins A and E affect acne condition? Clin Exp Dermatol 2006;31:430-434.
15. Amer M, Bahgat MR, Tosson Z, et al: Serum zinc in acne vulgaris. Int J Dermatol 1982;21:481-484.
Are you free from food addiction?
Are you free from hypertension medications and insulin?
Are you free from bypass surgery lurking around the corner?
Are you free from obesity and its accompanying diseases, aches and pains?
Are you free from the financial burdens of lost wages and expensive medical bills?
Eating a bowl of mixed greens & beans drizzled with a delicious nut-based dressing instead of a couple slices of pizza and can of soda may seem like a radical lifestyle change, but it really is a simple and cost effective solution to a very complicated problem; that being the loss of independence.
After the astronomical bills arrive in the mail who is going to pay them?
After the stroke who is going to tie the bib, feed, bathe, and even possibly change the diaper?
After degenerative disc disease has robbed mobility and peace, who is going to shop and prepare meals and soothe sleepless nights?
Paying the price today to be free from food addiction and accompanying diseases means enjoying the benefits of freedom on down-the-road.
I don’t know about you, but I want to climb sand dunes and watch sunsets shimmer on water, and produce my best creative works ever in my latter years. I want to laugh and play with my grandchildren and their children. I want to gracefully grow old; not rot in the prison of disease.
This Fourth of July, along with the festivities, fireworks, and parades; one of the best ways that we can honor those who have sacrificed their lives for our nation’s freedom is to commit or recommit to paying the price for our own personal freedoms!
Freedom now equals freedom on down-the-road.
It’s yours to enjoy!
Happy Fourth of July!
image credit: flickr by uhuru1701